Clinical Endocannabinoid Deficiency (CED / CECD) or
Clinical Endocannabinoid Deficiency Syndrome (CEDS)
Clinical Endocannabinoid Deficiency (CED / CECD) or Clinical Endocannabinoid Deficiency Syndrome (CEDS) is any condition or illness stemming from a lack of endocannabinoids in the body. In some cases, the body doesn’t naturally produce enough endocannabinoids or enough CB1 and CB2 receptors to sustain homeostasis. When homeostasis stalls, the body becomes unbalanced, and is vulnerable to disease.
Cannabinoid researcher Dr. Ethan Russo first hypothesized his theory of clinical endocannabinoid deficiency in Neuroendocrinology Letters in 2004. His theory proposed that, since the ECS plays a major role in homeostasis, it is possible that CECD may be related to a variety of ailments.
In 2016, Dr. Russo published a follow-up research review, stating that current research supported his initial theory. The research reveals that CECD may cause a hypersensitivity to pain, resulting in pain conditions such as migraines, fibromyalgia, and irritable bowel syndrome (IBS).
Each of these conditions has shown to be resistant to many standard treatments. Someone who has an endocannabinoid deficiency may suffer from one or more of these ailments due to their body’s inability to return to a balanced state.
Unfortunately, at this time, the only known ways to measure endocannabinoid levels are through invasive procedures that require samples from brain tissue. However, scientists are working on new, less invasive methods, such as analyzing saliva. There are also ways to self-diagnose CECD, based on several factors that include clinical diagnoses of chronic pain conditions, anxiety, and depression.
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Human Endocannabinoid System Receptors CB1 and CB2 are located throughout our brain and body. Optimal Health is acquired when these Receptors are communicating, or transmitting information
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